Soul of Success

Let’s Talk Soul Notes

Sermon Notes

Soul of Success: Key Thoughts

“Success is different than access “

Great people are looking for greatness
Great people want to stay great
Mediocre people can be misplaced by charismatic
Success is standing and endurance
Success is you being what you sold

“Fitness for duty = ready and available”

When you lend yourself to someone else, they own your body
What you think about you is how you portray you
When addiction cost more than pleasure you will get rid of it
The will, will make or break you
The will is a responder
It’s job is to see you live at peace with what you want
It constructs, concocts, forces and the like
Not every dream is prophetic
The heart will cause us to dream a lot of dreams
Dreams can show what we inseminated our soul for our will to go after
The will maneuvers situations that will engineer it
God made the soul to function for a reason
It’s not dealt with until it hits the planet
The soul is an earthly voyager

Relates to Genesis
He brooded on the waters
Brood = breeding
The Holy Spirit makes sure God’s word will “Be”
Everything starts coming out of the water
Moods can hatch things in your soul
Creativity does not mean we only create good things
We can create evil all day long
It’s easy to fix a prototype than it is to fix a population
Jesus is the prototype
He worked on Jesus and pass him on
Solomon says that man was made upright and he invented many schemes
Jesus designed your will to make your will happen
Answer and solutions give you power to correct
This world is desensitizing us to correction
When we don’t resolve things we become stuck
The will is waiting for instruction
Should not leave your soul at the mercy of someone’s will that is stronger than yours

Soul/Spirit/Body

The interface between your spirit and your body.
The soul is not partial or immaterial organ
The soul is a whole person that enables you to draw from your spirit to function in the physical world
The soul is the part of your make up that makes you equip for heaven or hell

Soul Detox = Nostalgia will track your detox process

When your body is rebelling against your soul you get emotional disturbance
It’s asking the soul to produce something it does not have
The soul goes into apothecary mode to create what your asking for
Addiction = the body opting out on life
The body is to metabolizing what is needed to keep you from feeling
The Euphoria is to keep you from feeling bad about being the wreck you are

What caused the body to ask for things it does not need:

Relationship
Temptation
Misdiagnosis
Fear
Insecurities
Ridicule/mockery and scorn

Key Thoughts:

Evil company corrupts good habits
Must decide if a person’s treatment of you should continue to feed your soul
You can become an infestation of cultural abuse
Saboteurs are childhood fantasies or edicts
We make decisions according to the court of your will
Your will procures what you want
Your will lays under your subconsciousness
It exposes itself when the circumstance or consequence appears
What drives and forms Judgments

Breaking Down the Soul Flow

The carotid artery causes the flow from head to heart or heart to head
The soul has the spirit
Heart has the mind
The mind has the body
The Flesh = the soul
The mind = subconsciousness
The Brain = conscious self
THe heart = architect
The will = engineer

Soul Organs:

We are made in the image and likeness of God
Nothing made he did not put himself through
Heart = Immaterial reservoir
Mind = Intelligence Order
Will = Emotional Commmander

*The soul can treat mental issues in the brain
The brain is smart and will create what it needs
Brain plasticity = Neural plasticity” redirects here. For journal, see Neural Plasticity (journal). For the 2014 Cold Specks album, see Neuroplasticity (album).
Neuroplasticity, also known as neural plasticity, or brain plasticity, is the ability of neural networks in the brain to change through growth and reorganization. These changes range from individual neuron pathways making new connections, to systematic adjustments like cortical remapping. Examples of neuroplasticity include circuit and network changes that result from learning a new ability, environmental influences, practice, and psychological stress.¹²³⁴⁵⁶

*Neuroplasticity was once thought by neuroscientists to manifest only during childhood,⁷⁸ but research in the latter half of the 20th century showed that many aspects of the brain can be altered (or are “plastic”) even through adulthood.⁹¹⁰ However, the developing brain exhibits a higher degree of plasticity than the adult brain.¹¹ Activity-dependent plasticity can have significant implications for healthy development, learning, memory, and recovery from brain damage.¹²¹³¹⁴ – Many neuroscientists used the term plasticity only to explain the regenerative capacity of the peripheral nervous system, which Cajal’s conceptual transfer of the term gave rise to a controversial discussion.²⁰ -

*Structural neuroplasticity
Structural plasticity is often understood as the brain’s ability to change its neuronal connections. New neurons are constantly produced and integrated into the central nervous system throughout the life span based on this type of neuroplasticity. Researchers nowadays use multiple cross-sectional imaging methods (i.e. magnetic resonance imaging (MRI), computerized tomography (CT)) to study the structural alterations of the human brains.³⁸ This type of neuroplasticity often studies the effect of various internal or external stimuli on the brain’s anatomical reorganization. The changes of grey matter proportion or the synaptic strength in the brain are considered as examples of structural neuroplasticity. Structural neuroplasticity is currently investigated more within the field of neuroscience in current academia.¹⁹

*Functional neuroplasticity
Functional plasticity refers to brain’s ability to alter and adapt the functional properties of neurons. The changes can occur in response to previous activity (activity-dependent plasticity) to acquire memory or in response to malfunction or damage of neurons (reactive plasticity) to compensate a pathological event. In the latter case the functions from one part of the brain transfer to another part of the brain based on the demand to produce recovery of behavioral or physiological processes.³⁹ Regarding physiological forms of activity-dependent plasticity, those involving synapses are referred to as synaptic plasticity. The strengthening or weakening of synapses that results in an increase or decrease of firing rate of the neurons are called long-term potentiation (LTP) and long-term depression (LTD), respectively, and they are considered as examples of synaptic plasticity that are associated with memory.⁴⁰ The cerebellum is a typical structure with combinations of LTP/LTD and redundancy within the circuitry, allowing plasticity at several sites.⁴¹ More recently it has become clearer that synaptic plasticity can be complemented by another form of activity-dependent plasticity involving the intrinsic excitability of neurons, which is referred to as intrinsic plasticity.⁴²⁴³ This, as opposed to homeostatic plasticity does not necessarily maintain the overall activity of a neuron within a network but contributes to encoding memories.⁴⁴ Also, many studies have been indicated functional neuroplasticity in the level of brain networks, where training alters the strength of functional connections.⁴⁵⁴⁶ Although a recent study discusses that these observed changes should not directly relate to neuroplasticity, since they may root in the systematic requirement of the brain network for reorganization.⁴⁷

*Applications and examples
The adult brain is not entirely “hard-wired” with fixed neuronal circuits. There are many instances of cortical and subcortical rewiring of neuronal circuits in response to training as well as in response to injury. There is evidence that neurogenesis (birth of brain cells) occurs in the adult, mammalian brain—and such changes can persist well into old age.¹⁰ The evidence for neurogenesis is mainly restricted to the hippocampus and olfactory bulb, but current research has revealed that other parts of the brain, including the cerebellum, may be involved as well.⁴⁸ However, the degree of rewiring induced by the integration of new neurons in the established circuits is not known, and such rewiring may well be functionally redundant.⁴⁹

*There is now ample evidence[citation needed] for the active, experience-dependent re-organization of the synaptic networks of the brain involving multiple inter-related structures including the cerebral cortex. The specific details of how this process occurs at the molecular and ultrastructural levels are topics of active neuroscience research. The way experience can influence the synaptic organization of the brain is also the basis for a number of theories of brain function including the general theory of mind and neural Darwinism. The concept of neuroplasticity is also central to theories of memory and learning that are associated with experience-driven alteration of synaptic structure and function in studies of classical conditioning in invertebrate animal models such as Aplysia.

*Treatment of brain damage
A surprising consequence of neuroplasticity is that the brain activity associated with a given function can be transferred to a different location; this can result from normal experience and also occurs in the process of recovery from brain injury. Neuroplasticity is the fundamental issue that supports the scientific basis for treatment of acquired brain injury with goal-directed experiential therapeutic programs in the context of rehabilitation approaches to the functional consequences of the injury.

*Neuroplasticity is gaining popularity as a theory that, at least in part, explains improvements in functional outcomes with physical therapy post-stroke. Rehabilitation techniques that are supported by evidence which suggest cortical reorganization as the mechanism of change include constraint-induced movement therapy, functional electrical stimulation, treadmill training with body-weight support, and virtual reality therapy. Robot assisted therapy is an emerging technique, which is also hypothesized to work by way of neuroplasticity, though there is currently insufficient evidence to determine the exact mechanisms of change when using this method.⁵⁰

*One group has developed a treatment that includes increased levels of progesterone injections in brain-injured patients. “Administration of progesterone after traumatic brain injury⁵¹ (TBI) and stroke reduces edema, inflammation, and neuronal cell death, and enhances spatial reference memory and sensory-motor recovery.”⁵² In a clinical trial, a group of severely injured patients had a 60% reduction in mortality after three days of progesterone injections.⁵³ However, a study published in the New England Journal of Medicine in 2014 detailing the results of a multi-center NIH-funded phase III clinical trial of 882 patients found that treatment of acute traumatic brain injury with the hormone progesterone provides no significant benefit to patients when compared with placebo.⁵⁴

*A diagrammatic explanation of the mirror box. The patient places the intact limb into one side of the box (in this case the right hand) and the amputated limb into the other side. Due to the mirror, the patient sees a reflection of the intact hand where the missing limb would be (indicated in lower contrast). The patient thus receives artificial visual feedback that the “resurrected” limb is now moving when they move the good hand.
Main articles: Phantom limb and Mirror box
In the phenomenon of phantom limb sensation, a person continues to feel pain or sensation within a part of their body that has been amputated. This is strangely common, occurring in 60–80% of amputees.⁵⁸ An explanation for this is based on the concept of neuroplasticity, as the cortical maps of the removed limbs are believed to have become engaged with the area around them in the postcentral gyrus. This results in activity within the surrounding area of the cortex being misinterpreted by the area of the cortex formerly responsible for the amputated limb.

*The relationship between phantom limb sensation and neuroplasticity is a complex one. In the early 1990s V.S. Ramachandran theorized that phantom limbs were the result of cortical remapping. However, in 1995 Herta Flor and her colleagues demonstrated that cortical remapping occurs only in patients who have phantom pain.⁵⁹ Her research showed that phantom limb pain (rather than referred sensations) was the perceptual correlate of cortical reorganization.⁶⁰ This phenomenon is sometimes referred to as maladaptive plasticity.

*In 2009, Lorimer Moseley and Peter Brugger carried out an experiment in which they encouraged arm amputee subjects to use visual imagery to contort their phantom limbs into impossible[clarification needed] configurations. Four of the seven subjects succeeded in performing impossible movements of the phantom limb. This experiment suggests that the subjects had modified the neural representation of their phantom limbs and generated the motor commands needed to execute impossible movements in the absence of feedback from the body.⁶¹ The authors stated that: “In fact, this finding extends our understanding of the brain’s plasticity because it is evidence that profound changes in the mental representation of the body can be induced purely by internal brain mechanisms—the brain truly does change itself.”

*ain article: Chronic pain
Individuals who suffer from chronic pain experience prolonged pain at sites that may have been previously injured, yet are otherwise currently healthy. This phenomenon is related to neuroplasticity due to a maladaptive reorganization of the nervous system, both peripherally and centrally. During the period of tissue damage, noxious stimuli and inflammation cause an elevation of nociceptive input from the periphery to the central nervous system. Prolonged nociception from the periphery then elicits a neuroplastic response at the cortical level to change its somatotopic organization for the painful site, inducing central sensitization.⁶² For instance, individuals experiencing complex regional pain syndrome demonstrate a diminished cortical somatotopic representation of the hand contralaterally as well as a decreased spacing between the hand and the mouth.⁶³ Additionally, chronic pain has been reported to significantly reduce the volume of grey matter in the brain globally, and more specifically at the prefrontal cortex and right thalamus.⁶⁴ However, following treatment, these abnormalities in cortical reorganization and grey matter volume are resolved, as well as their symptoms. Similar results have been reported for phantom limb pain,⁶⁵ chronic low back pain⁶⁶ and carpal tunnel syndrome.⁶⁷

*Meditation
Main article: Research on meditation
A number of studies have linked meditation practice to differences in cortical thickness or density of gray matter.⁶⁸⁶⁹⁷⁰⁷¹ One of the most well-known studies to demonstrate this was led by Sara Lazar, from Harvard University, in 2000.⁷² Richard Davidson, a neuroscientist at the University of Wisconsin, has led experiments in collaboration with the Dalai Lama on effects of meditation on the brain. His results suggest that long-term or short-term practice of meditation can lead to different levels of activities in brain regions associated with effects such as attention, anxiety, depression, fear, anger, and compassion as well as the ability of the body to heal itself. These functional changes may be caused by changes in the physical structure of the brain.⁷³⁷⁴⁷⁵⁷⁶

*Fitness and exercise
See also: Neurobiological effects of physical exercise § Structural growth
Aerobic exercise promotes adult neurogenesis by increasing the production of neurotrophic factors (compounds that promote growth or survival of neurons), such as brain-derived neurotrophic factor (BDNF), insulin-like growth factor 1 (IGF-1), and vascular endothelial growth factor (VEGF).⁷⁷⁷⁸⁷⁹ Exercise-induced neurogenesis in the hippocampus is associated with measurable improvements in spatial memory.⁸⁰⁸¹⁸²⁸³ Consistent aerobic exercise over a period of several months induces marked clinically significant improvements in executive function (i.e., the “cognitive control” of behavior) and increased gray matter volume in multiple brain regions, particularly those that give rise to cognitive control.⁷⁹⁸⁰⁸⁴⁸⁵ The brain structures that show the greatest improvements in gray matter volume in response to aerobic exercise are the prefrontal cortex and hippocampus;⁷⁹⁸⁰⁸¹ moderate improvements are seen in the anterior cingulate cortex, parietal cortex, cerebellum, caudate nucleus, and nucleus accumbens.⁷⁹⁸⁰⁸¹ Higher physical fitness scores (measured by VO2 max) are associated with better executive function, faster processing speed, and greater volume of the hippocampus, caudate nucleus, and nucleus accumbens.⁸⁰

*Deaf individuals have enhanced peripheral visual attention,⁸⁹ better motion change but not color change detection ability in visual tasks,⁸⁷⁸⁸⁹⁰ more effective visual search,⁹¹ and faster response time for visual targets⁹²⁹³ compared to hearing individuals. Altered visual processing in deaf people is often found to be associated with the repurposing of other brain areas including primary auditory cortex, posterior parietal association cortex (PPAC), and anterior cingulate cortex (ACC).⁹⁴ A review by Bavelier et al. (2006) summarizes many aspects on the topic of visual ability comparison between deaf and hearing individuals.⁹⁵

*Neuroplasticity is involved in the development of sensory function. The brain is born immature and then adapts to sensory inputs after birth. In the auditory system, congenital hearing loss, a rather frequent inborn condition affecting 1 of 1000 newborns, has been shown to affect auditory development, and implantation of a sensory prostheses activating the auditory system has prevented the deficits and induced functional maturation of the auditory system.⁹⁸ Due to a sensitive period for plasticity, there is also a sensitive period for such intervention within the first 2–4 years of life. Consequently, in prelingually deaf children, early cochlear implantation, as a rule, allows the children to learn the mother language and acquire acoustic communication.⁹⁹

*Blindness
Due to vision loss, the visual cortex in blind people may undergo cross-modal plasticity, and therefore other senses may have enhanced abilities. Or the opposite could occur, with the lack of visual input weakening the development of other sensory systems. One study suggests that the right posterior middle temporal gyrus and superior occipital gyrus reveal more activation in the blind than in the sighted people during a sound-moving detection task.¹⁰⁰ Several studies support the latter idea and found weakened ability in audio distance evaluation, proprioceptive reproduction, threshold for visual bisection, and judging minimum audible angle.¹⁰¹¹⁰²

*Human echolocation is a learned ability for humans to sense their environment from echoes. This ability is used by some blind people to navigate their environment and sense their surroundings in detail. Studies in 2010¹⁰³ and 2011¹⁰⁴ using functional magnetic resonance imaging techniques have shown that parts of the brain associated with visual processing are adapted for the new skill of echolocation. Studies with blind patients, for example, suggest that the click-echoes heard by these patients were processed by brain regions devoted to vision rather than audition.¹⁰⁴

*MRI studies of 1713 participants shows that both children and adults with attention deficit hyperactivity disorder (ADHD) have smaller volumes of the nucleus accumbens, amygdala, caudate, hippocampus, putamen, and overall cortical and intracranial volume; and have less surface area and cortical thickness, compared to people without ADHD.¹⁰⁵¹⁰⁶ Brain volume does not correlate to intelligence, or intelligence quotient (IQ).¹⁰⁷ People with ADHD exhibit atypical neuroconnectivity. In particular, it has been hypothesized that ADHD symptomatology may arise from a deviation from neurotypical synchronization and interaction within and between these large-scale networks during brain development. Investigating functional connectivity using the sub-second temporal resolution of electroencephalography (EEG) instead allows for the measurement of a wider range of brain oscillatory phenomena, including transient changes in connectivity during cognition and behavior.¹⁰⁸

*Neuroplasticity is most active in childhood as a part of normal human development, and can also be seen as an especially important mechanism for children in terms of risk and resiliency.¹¹³ Trauma is considered a great risk as it negatively affects many areas of the brain and puts a strain on the sympathetic nervous system from constant activation. Trauma thus alters the brain’s connections such that children who have experienced trauma may be hyper vigilant or overly aroused.¹¹⁴ However, a child’s brain can cope with these adverse effects through the actions of neuroplasticity.¹¹⁵

*There are many examples of neuroplasticity in human development. For example, Justine Ker and Stephen Nelson looked at the effects of musical training on neuroplasticity, and found that musical training can contribute to experience dependent structural plasticity. This is when changes in the brain occur based on experiences that are unique to an individual. Examples of this are learning multiple languages, playing a sport, doing theatre, etc. A study done by Hyde in 2009, showed that changes in the brain of children could be seen in as little as 15 months of musical training.¹¹⁷ Ker and Nelson suggest this degree of plasticity in the brains of children can “help provide a form of intervention for children… with developmental disorders and neurological diseases.”¹¹⁸

*Within the class Aves, black-capped chickadees experience an increase in the volume of their hippocampus and strength of neural connections to the hippocampus during fall months.¹²⁶¹²⁷ These morphological changes within the hippocampus which are related to spatial memory are not limited to birds, as they can also be observed in rodents and amphibians.¹²³ In songbirds, many song control nuclei in the brain increase in size during mating season.¹²³ Among birds, changes in brain morphology to influence song patterns, frequency, and volume are common.¹²⁸ Gonadotropin-releasing hormone (GnRH) immunoreactivity, or the reception of the hormone, is lowered in European starlings exposed to longer periods of light during the day.¹¹⁹¹²⁰

Traumatic brain injury research
Randy Nudo’s group found that if a small stroke (an infarction) is induced by obstruction of blood flow to a portion of a monkey’s motor cortex, the part of the body that responds by movement moves when areas adjacent to the damaged brain area are stimulated. In one study, intracortical microstimulation (ICMS) mapping techniques were used in nine normal monkeys. Some underwent ischemic-infarction procedures and the others, ICMS procedures. The monkeys with ischemic infarctions retained more finger flexion during food retrieval and after several months this deficit returned to preoperative levels.¹³⁰ With respect to the distal forelimb representation, “postinfarction mapping procedures revealed that movement representations underwent reorganization throughout the adjacent, undamaged cortex.”¹³⁰ Understanding of interaction between the damaged and undamaged areas provides a basis for better treatment plans in stroke patients. Current research includes the tracking of changes that occur in the motor areas of the cerebral cortex as a result of a stroke. Thus, events that occur in the reorganization process of the brain can be ascertained. Nudo is also involved in studying the treatment plans that may enhance recovery from strokes, such as physiotherapy, pharmacotherapy, and electrical-stimulation therapy.

Jon Kaas, a professor at Vanderbilt University, has been able to show “how somatosensory area 3b and ventroposterior (VP) nucleus of the thalamus are affected by longstanding unilateral dorsal-column lesions at cervical levels in macaque monkeys.”¹³¹ Adult brains have the ability to change as a result of injury but the extent of the reorganization depends on the extent of the injury. His recent research focuses on the somatosensory system, which involves a sense of the body and its movements using many senses. Usually, damage of the somatosensory cortex results in impairment of the body perception. Kaas’ research project is focused on how these systems (somatosensory, cognitive, motor systems) respond with plastic changes resulting from injury.¹³¹

One recent study of neuroplasticity involves work done by a team of doctors and researchers at Emory University, specifically Dr. Donald Stein¹³² and Dr. David Wright. This is the first treatment in 40 years that has significant results in treating traumatic brain injuries while also incurring no known side effects and being cheap to administer.⁵³ Dr. Stein noticed that female mice seemed to recover from brain injuries better than male mice, and that at certain points in the estrus cycle, females recovered even better. This difference may be attributed to different levels of progesterone, with higher levels of progesterone leading to the faster recovery from brain injury in mice. However, clinical trials showed progesterone offers no significant benefit for traumatic brain injury in human patients.¹³³

Aging
Transcriptional profiling of the frontal cortex of persons ranging from 26 to 106 years of age defined a set of genes with reduced expression after age 40, and especially after age 70.¹³⁴ Genes that play central roles in synaptic plasticity were the most significantly affected by age, generally showing reduced expression over time. There was also a marked increase in cortical DNA damage, likely oxidative DNA damage, in gene promoters with aging.¹³⁴

Reactive oxygen species appear to have a significant role in the regulation of synaptic plasticity and cognitive function.¹³⁵ However age-related increases in reactive oxygen species may also lead to impairments in these functions.

Multilingualism
The beneficial effect of multilingualism on people’s behavior and cognition is well-known nowadays. Numerous studies have shown that people who study more than one language have better cognitive functions and flexibilities than people who only speak one language. Bilinguals are found to have longer attention spans, stronger organization and analyzation skills, and a better theory of mind than monolinguals. Researchers have found that the effect of multilingualism on better cognition is due to neuroplasticity.

In one prominent study, neurolinguists used a voxel-based morphometry (VBM) method to visualize the structural plasticity of brains in healthy monolinguals and bilinguals. They first investigated the differences in density of grey and white matter between two groups and found the relationship between brain structure and age of language acquisition. The results showed that grey-matter density in the inferior parietal cortex for multilinguals were significantly greater than monolinguals. The researchers also found that early bilinguals had a greater density of grey matter relative to late bilinguals in the same region. The inferior parietal cortex is a brain region highly associated with the language learning, which corresponds to the VBM result of the study.¹³⁶

Recent studies have also found that learning multiple languages not only re-structures the brain but also boosts brain’s capacity for plasticity. A recent study found that multilingualism not only affects the grey matter but also white matter of the brain. White matter is made up of myelinated axons that is greatly associated with learning and communication. Neurolinguists used a diffusion tensor imaging (DTI) scanning method to determine the white matter intensity between monolinguals and bilinguals. Increased myelinations in white matter tracts were found in bilingual individuals who actively used both languages in everyday life. The demand of handling more than one language requires more efficient connectivity within the brain, which resulted in greater white matter density for multilinguals.¹³⁷

While it is still debated whether these changes in brain are result of genetic disposition or environmental demands, many evidences suggest that environmental, social experience in early multilinguals affect the structural and functional reorganization in the brain.¹³⁸¹³⁹

-Learn to stop sabotaging your success

To revoke codes that keep you sabotaging your success
To reprogram the panic and defense network you use to protect you sabotage your success
To expire the emotional commands you trigger in ion order to sabotage your success
Replace your comment o sabotage with a resolve to succeed

Must examine the season of life we are most vulnerable and susceptible to sabotage

Blood is the life of all flesh
Spirit life traits life to the soul
Spirit life transits life to the soul
The soul is an interferance
The body and soul are considered a unit
The soul produces blood
The soul distributes blood chemistry to the body
The natural blood and spirit blood hot the same
Spirit generates life to the soul
Soul distributes it to the flesh
Blood determines bloodline
Blood chemistry animates and sustains the body
Soul send blood to liver
Spirit has one law
Soul has another law
The law of the spirit is life, the law of the soul is death
Spirit law is eternal and immortal
%(color-red)Soul law = carnal and mortal %
Spirit life and laws cam through Jesus Christ
Why we must be born again from above
The first man Adam was made a living soul
The last Adam was made a quickening spirit

Spirit life = fire (energy)
Soul life = fluid life

The spirit masters the soul
The body without spirit is dead
Soul and spirit = 1 unit
God can separate soul and spirit
(Jesus did it on the cross)

Table Talk – Plant Soul Thoughts
Rehashing
Concealed Reactions
Emotional Soothing
Consensus Agreement
Unspoken Opinions
Fears doubts
Plots & Schemes
Reactions and retaliations
Secret heart vows

We are not in bondage because God cannot set us free
We cannot think we don’t play a volitional role in it
Soul contracts are real
Satan cannot just bind you at His will as a child of God
Israel was elevated about the curse of the law by contract
Ie: Blessings or cursings
We must live out the life of the contract on our behavior when violate it
God brings deliverance when a new generation is born that was not under that contract (or sentence)

God’s word is upheld by us